![]() Not much is known about flubromazepam or its metabolites. I have read that this drug is highly selective to the α1 an α5 sub units of the GABA-A receptor (hypnotic sub units). I find myself nodding off at work, while applying for new jobs, while on the subway. I was finding it very difficult to function on flubromazepam in lieu of the sedation. Here are my insights and reasons for switching: But after 8 months of trying to wean down with flubromazepam, I had ended up in severe tolerance withdrawal and blew my taper. I had previously successfully cut a clonazolam addiction with 7 months of diclazepam tapering. At the time, my understanding of tapering was basic the longer the half life, the better the drug is for tapering. After my relapse February, I had decided that to try fpam for a taper.
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